wabmester wrote: »

@totaloblivia, that last "heat map" in the image 2H gives an idea of why we differ. For example, if you have high HDL, you have an improved glycemic response to everything except fructose. High triglycerides are associated with a worse glycemic response to all 4 of those test meals.

They looked at a bunch of factors, and none of them really stood out as THE factor, but combining them all into an algorithm gives them a pretty good predictor (R=0.7).

They also looked the effects of a "good" and "bad" diet on the microbiome. I think @DittoDan is trying to do some microbiome self-experimenting. There's a good summary here:

Bifidobacterium adolescentis, for which low levels were reported to be associated with greater weight loss (Santacruz et al., 2009), generally decrease in RA following the “good” diet and increase following the “bad” diet (Figure 6C,D). Similarly, TIIDM has been associated with low levels of Roseburia inulinivorans (Qin et al., 2012; Figure 6E), Eubacterium eligens (Karlsson et al., 2013), and Bacteroides vulgatus (Ridaura et al., 2013), and all these bacteria increase following the “good” diet and decrease following the “bad” diet (Figure 6C). The Bacteroidetes phylum, for which low levels associate with obesity and high fasting glucose (Turnbaugh et al., 2009), increases following the “good” diet and decreases following the “bad” diet (Figure 6C). Low levels of Anaerostipes associate with improved glucose tolerance and reduced plasma triglyceride levels in mice (Everard et al., 2011) and indeed these bacteria decrease following the “good” diet and increase following the “bad” diet (Figure 6C). Finally, low levels of Alistipes putredinis associate with obesity (Ridaura et al., 2013) and this bacteria increased following the “good” diet (Figure 6C).