Vegetable oils... Friend or Foe?

So assuming vegetable oils are bad, then..... vegetables are bad? Avocados etc should be avoided? Olives?

So assuming vegetable oils are bad, then..... vegetables are bad? Avocados etc should be avoided? Olives?

its not that, there are some studies that talk about how polyunsaturated fat is linked with pro-inflammation effects on the heart. Which is the true cause of heart disease.

However, many of the studies are still inconclusive with other studies so its very skeptical. Avocado is monounsaturated fat.

Avocado is a fruit btw

So assuming vegetable oils are bad, then..... vegetables are bad? Avocados etc should be avoided? Olives?

its not that, there are some studies that talk about how polyunsaturated fat is linked with pro-inflammation effects on the heart. Which is the true cause of heart disease.

However, many of the studies are still inconclusive with other studies so its very skeptical. Avocado is monounsaturated fat.

Avocado is a fruit btw

'Vegetable oil" is generally made from a variety of seed oils. So it's not really "oil from vegetables."

Avocado oil is considered a vegetable oil, just like canola oil, peanut oil, corn oil, and soybean oil - none of which are made from vegetables.

regardless, the studies are based off of high intake of PUFA, which is something many processed foods are based off of.

However people keep talking about heart disease, etc. You never hear about someone who is in shape and that eats garbage getting heart disease pre 65 unless its some rare case. The heart is working 24/7 and people seem to be surprised they dont live forever

So assuming vegetable oils are bad, then..... vegetables are bad? Avocados etc should be avoided? Olives?

I think he means in terms of cooking and such. Solvent extracted vs. cold pressed, etc.

That's fine. I'd like to see the studies. If the discussion is PUFA vs other oils as opposed to "vegetable oils" vs other oils that would make more sense.

Now that you mention it, PUFA is more along the lines of what I meant.

Abstract
BACKGROUND:
Replacing SFAs with vegetable PUFAs has cardiometabolic benefits, but the effects on liver fat are unknown. Increased dietary n-6 PUFAs have, however, also been proposed to promote inflammation-a yet unproven theory.
OBJECTIVE:
We investigated the effects of PUFAs on liver fat, systemic inflammation, and metabolic disorders.
DESIGN:
We randomly assigned 67 abdominally obese subjects (15% had type 2 diabetes) to a 10-wk isocaloric diet high in vegetable n-6 PUFA (PUFA diet) or SFA mainly from butter (SFA diet), without altering the macronutrient intake. Liver fat was assessed by MRI and magnetic resonance proton (1H) spectroscopy (MRS). Proprotein convertase subtilisin/kexin type-9 (PCSK9, a hepatic LDL-receptor regulator), inflammation, and adipose tissue expression of inflammatory and lipogenic genes were determined.
RESULTS:
A total of 61 subjects completed the study. Body weight modestly increased but was not different between groups. Liver fat was lower during the PUFA diet than during the SFA diet [between-group difference in relative change from baseline; 16% (MRI; P < 0.001), 34% (MRS; P = 0.02)]. PCSK9 (P = 0.001), TNF receptor-2 (P < 0.01), and IL-1 receptor antagonist (P = 0.02) concentrations were lower during the PUFA diet, whereas insulin (P = 0.06) tended to be higher during the SFA diet. In compliant subjects (defined as change in serum linoleic acid), insulin, total/HDL-cholesterol ratio, LDL cholesterol, and triglycerides were lower during the PUFA diet than during the SFA diet (P < 0.05). Adipose tissue gene expression was unchanged.
CONCLUSIONS:
Compared with SFA intake, n-6 PUFAs reduce liver fat and modestly improve metabolic status, without weight loss. A high n-6 PUFA intake does not cause any signs of inflammation or oxidative stress. Downregulation of PCSK9 could be a novel mechanism behind the cholesterol-lowering effects of PUFAs. This trial was registered at clinicaltrials.gov as NCT01038102.



It is mainly many paleo people that talk about how bad PUFA is. I cant even remember where to find the study that says its bad for you.

That's fine. I'd like to see the studies. If the discussion is PUFA vs other oils as opposed to "vegetable oils" vs other oils that would make more sense.

Now that you mention it, PUFA is more along the lines of what I meant.

Abstract
BACKGROUND:
Replacing SFAs with vegetable PUFAs has cardiometabolic benefits, but the effects on liver fat are unknown. Increased dietary n-6 PUFAs have, however, also been proposed to promote inflammation-a yet unproven theory.
OBJECTIVE:
We investigated the effects of PUFAs on liver fat, systemic inflammation, and metabolic disorders.
DESIGN:
We randomly assigned 67 abdominally obese subjects (15% had type 2 diabetes) to a 10-wk isocaloric diet high in vegetable n-6 PUFA (PUFA diet) or SFA mainly from butter (SFA diet), without altering the macronutrient intake. Liver fat was assessed by MRI and magnetic resonance proton (1H) spectroscopy (MRS). Proprotein convertase subtilisin/kexin type-9 (PCSK9, a hepatic LDL-receptor regulator), inflammation, and adipose tissue expression of inflammatory and lipogenic genes were determined.
RESULTS:
A total of 61 subjects completed the study. Body weight modestly increased but was not different between groups. Liver fat was lower during the PUFA diet than during the SFA diet [between-group difference in relative change from baseline; 16% (MRI; P < 0.001), 34% (MRS; P = 0.02)]. PCSK9 (P = 0.001), TNF receptor-2 (P < 0.01), and IL-1 receptor antagonist (P = 0.02) concentrations were lower during the PUFA diet, whereas insulin (P = 0.06) tended to be higher during the SFA diet. In compliant subjects (defined as change in serum linoleic acid), insulin, total/HDL-cholesterol ratio, LDL cholesterol, and triglycerides were lower during the PUFA diet than during the SFA diet (P < 0.05). Adipose tissue gene expression was unchanged.
CONCLUSIONS:
Compared with SFA intake, n-6 PUFAs reduce liver fat and modestly improve metabolic status, without weight loss. A high n-6 PUFA intake does not cause any signs of inflammation or oxidative stress. Downregulation of PCSK9 could be a novel mechanism behind the cholesterol-lowering effects of PUFAs. This trial was registered at clinicaltrials.gov as NCT01038102.



It is mainly many paleo people that talk about how bad PUFA is. I cant even remember where to find the study that says its bad for you.

There's plenty of studies out there...I could cherry pick but I'm not really interested, sorry. Ray Peat has plenty listed on his website.
http://raypeat.com/articles/articles/unsaturatedfats.shtml