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ebonymph18 · October 2015

Let's see where we end up!

JustMissTracy · October 2015

Gala Garden, Hot Apple Pie, Sassy Citrus and Very Berry Cherry are missing

Unknown · October 2015

This content has been removed.

_incogNEATo_ · October 2015

Cambria76 · October 2015

Just getting on plane. Had to wait 40 min for the crew they were late.

SugarBabyGirl · October 2015

FabulousFantasticFifty · October 2015

http://www.angieslist.com/companylist/us/co/colorado-springs/the-heat-depot,-llc-reviews-7949923.htm

myheartsabattleground · October 2015

SugarBabyGirl wrote: »

OUCH !

JustMissTracy · October 2015

That guy is an absolute jerk...and what's with his face? Is he sick or something?

moijo1 · October 2015

sorry but you asked:-

Intracranial pressure (ICP) remains one of the principal treatment targets for traumatic brain injury (TBI) (1). Elevation of ICP is due to brain edema, mass effect from hemorrhagic lesions, and possibly from disrupted pressure autoregulation. The treatments for ICP have been focused on these pathophysiologic mechanisms. While the treatment principals for elevated ICP have been codified into guidelines and practiced by many intensivists, ICP often remains refractory to treatment. Indeed, treatment failure resulting in herniation and death occurs frequently. The mechanisms for persistently elevated ICP have not been well elucidated, but candidate mechanisms include sustained hyperemia, excitotoxicity, and osmotic rebound. In contrast, little attention has been paid to the adverse effects of epileptic activity upon the extent or duration of elevated ICP after TBI.

In recent years, post-traumatic seizures have been documented to occur frequently after human TBI (2). Indeed, seizures are frequent after a variety of hemorrhagic injuries to the brain (3, 4). The incidence of seizures increases as a function of the severity of injury, and other selected features of the injury such as the presence of hemorrhagic contusions. Post-traumatic seizures previously have been associated with increased glycolysis in both animal and human metabolic imaging studies (5, 6) and have been associated with alterations of brain levels of glutamate and other excitatory amino acids (7). However, it is presently unclear whether post-traumatic seizures result in an increase in ICP or other adverse consequences for the patient. This has resulted in uncertainty about how aggressively to treat seizures, especially those that are nonconvulsive or purely electrographic.

It is in this context that we studied the cerebral hemodynamic and metabolic effects of electrographic post-traumatic seizures in a cohort of TBI patients monitored by continuous electroencephalograms (EEGs). The primary hypothesis was that nonconvulsive electrographic seizures result in an episodic increase in ICP. The secondary hypothesis was that electrographic seizures result in an increase in microdialysis lactate/pyruvate ratio (LPR). We utilized a within-subjects, before-after seizures design as well as a separate between-subjects cohort comparison design to test our hypotheses.

TUsphg8B4zrapVx · October 2015

Starting Weight (SW): 240
Challenge Starting Weight (CSW): 240
Current Weight (CW): 210
Ultimate Goal Weight (UGW): 139

OneHundredToLose · October 2015

add_action('pre_get_posts', 'ad_filter_categories');
function ad_filter_categories($query) {
if ($query->is_main_query() && is_home()) {
$query->set('category_name','blog, uncategorized');
}
}

I'm at work, needless to say...

MrsBooBear · October 2015

http://s3-media4.ak.yelpcdn.com/bphoto/dpxUt6yj5yKqYNAD5Rz6rg/o.jpg

pie_eyes · October 2015

Beaverhausen

TheRoadDog · October 2015

While this doesn’t accurately reflect all of the tonnage that we are behind on, it is three major contributors. Not only for the tonnage they represent, but for the tonnage they displace as we smooth them out.

I’ve also attached today’s Past Due to Schedule, which represented 16.8 Tons.