Am I type 2 diabetic? WTF is wrong???Please help!!!!

Diabetes is high blood sugar, not low. But those do sound like low numbers, it could be hypoglycemia.

GO TO THE DOCTOR. There are so many things besides diabetes that can cause vision episodes...Blurred vision is one of the primary initial symptoms of MS. Maybe it isn't the sugar. Don't get diagnosed by the internet!

nvmomketo wrote: »

butteryflapjacks wrote: »

I need help. Obviously,I need a medical opinion but for now some solid help would be appreciated.
I'm 6'1,170-175lbs,workout consistently but over the past 2 years I've experienced weird episodes where I lose my vision(blurry) and ONLY drinking sugar cures this(takes about 15 minutes). I've been testing my blood sugar and my fasting blood sugar is always in the 70s,my post meal(1-2 hours) is 70s,high 60s,and 1 time it was 85. What does this mean? How could my blood sugar literally never spike? I've purposely ate HIGH simple carb meals just for the testing and still have yet to have a reading over 90 no matter when I test. So much confusion and any help would be appreciated....Thanks.

Sounds like reactive hypoglycemia to me, which can be an indication of insulin resistance. Those with reactive hypoglycemia will have a normal to high BG spike (about 1 hour) after eating and then their BG drops to below where it started. The fast drop in BG causes (false) hypoglycemic symptoms due to the large drop and not because you are actually hypoglycemic. I used to get light headed and tunnel vision, weak and tremors about 2 hours after eating. It felt like a low but I was still in the 70s or 80s, although an hour after eating it was probably into the 100s.

Eating low carb has completely fixed the situation for me. I can't remember the last time I had false hypoglycemic symptoms, whereas I used to have symptoms a couple of times per day.

This!!! I experienced exact same thing. Could not get my BS over 100 at all. Diagnosed with Reactive Hypoglycemia and eventually Insulin Resistance (IR). My IR has progressed that I now take Metformin and my BS does go over 100 (even fasting). I started low carb several months ago and have seen very positive changes in my blood sugar.

For some clarification, because there's some conflicting advice on T1 versus T2 diabetes:

Type 1A diabetes (90% of type 1 diabetes is type 1A) is a result of an autoimmune attack on the insulin-producing beta cells of the pancreas (people recently diagnosed with T1ADM will be positive for certain autoantibodies, like GAD-65). Current theories suggest that there is a genetic predisposition for it (particularly within the HLA antigens on chromosome 6 -possessing certain alleles of these antigens increases one's risks for autoimmune diseases like T1ADM, MS, lupus, etc. If an immediate family member has T1DM, the risk of someone else in the family getting T1DM increases 10-fold) on top of being exposed to an environmental trigger that "kicks off" the autoimmune attack (some research suggests it might be a viral trigger). Besides being unable to produce insulin (and amylin), the pancreas continues to function normally (it can still produce glucagon, somatostatin, pancreatic polypeptide, digestive enzymes, etc.). The autoimmune attack can happen at any time (it happened to me at 18, and I have met other persons with T1D who got it in their 60s), which is why it previously being called juvenile diabetes is inaccurate. Type 1B diabetes is idiopathic diabetes (they essentially depend on insulin for survival, but they're negative for autoantibodies seen in type 1A/LADA or have other pathologies like pancreatitis or a pancreatectomy that would make them not produce insulin).

Type 2 diabetes's main hallmark is insulin resistance. Insulin resistance is associated with weight/body composition (muscle is really insulin sensitive, while fat increases insulin resistance), heredity (one's risk of T2DM increases if you have an immediate relative with it), certain medications (corticosteroids is a big one), certain medical conditions (PCOS), and lifestyle factors (physical activity, diet, etc), but many physically fit/active people go on to develop T2DM (or actually end up having LADA or a slow-acting version of T1DM, but that is another story). Unlike with type 1 diabetes, type 2 diabetics initially produce very large quantities of insulin (because of the insulin resistance). Over time, the beta cells of the pancreas may become "burned out", which might warrant a person with type 2 diabetes to need to go on insulin (they're not a type 1 diabetic, because there was no autoimmune attack present).