Egg Fanatic

liftingbro

There are people who can eat anything they want and they will have a normal cholesterol and there will be others who will have a high cholesterol no matter what they eat or what medications they are on. Because of this variation in lipid metabolism, it is imparative to have compared differing diet's affects on cholesterol levels on an individaul basis.

Not trying to argue with ya man but think about this paragraph you wrote. People that can eat anything they want and have normal cholesterol, and then others who will have high cholesterol no matter what they eat. Those with the high cholesterol levels most likely (not always) are hereditary or another cause, but it's MOST LIKELY not from eating a certain type of food bro.

I already said there is a geneitc component to lipid metabolism. This is part of the reason why the study needed to be done on a individual basis with varied diet. However, most people who eat too much cholesterol/fatty foods will have high cholesterol levels.The interesting thing here would be to find out why, specifically, some people can metabolize lipids very well while others cannot. It does appear that the more obese you are the less easy it is to metabolize lipids, so that's one known angle but then there are also some thin people who struggle with cholesterol levels.

We do know that for most people increased lipid intake results in increased cholesterol levels. There is some evidance that obese people are less efficient in metabolizing lipids (so more likely to struggle with cholesterol levels) but at this point the rest of the exceptions are not well enough studied to know for sure how they work.

So, what I'm saying is it's not correct to say that dietary cholestrol does not affect serum cholesterol levels because for the vast majority of people it does. When I worked in chemistry on a daily basis I saw the effects of lipid intake directly on lipid levels, this is a known fact. However, not everyone metabolizes lipids at the same level of efficiency. I will bet anyone $1000 that if you go eat a double cheese burger with bacon and a few hours later get your lipids done they will be completely eff'd compared to your fasting lipids.

joejccva71

Not to beat the dead horse, but these were quotes that Alan Aragon used himself.

Fernandez ML. Dietary cholesterol provided by eggs and plasma lipoproteins in healthy populations. Curr Opin Clin Nutr Metab Care. 2006 Jan;9(1):8-12.

PURPOSE OF REVIEW: Extensive research has not clearly established a link between egg consumption and risk for coronary heart disease. The effects of egg intake on plasma lipids and low-density lipoprotein (LDL) atherogenicity in healthy populations need to be addressed. RECENT FINDINGS: The lack of connection between heart disease and egg intake could partially be explained by the fact that dietary cholesterol increases the concentrations of both circulating LDL and high-density lipoprotein (HDL) cholesterol in those individuals who experience an increase in plasma cholesterol following egg consumption (hyperresponders). It is also important to note that 70% of the population experiences a mild increase or no alterations in plasma cholesterol concentrations when challenged with high amounts of dietary cholesterol (hyporesponders). Egg intake has been shown to promote the formation of large LDL, in addition to shifting individuals from the LDL pattern B to pattern A, which is less atherogenic. Eggs are also good sources of antioxidants known to protect the eye; therefore, increased plasma concentrations of lutein and zeaxanthin in individuals consuming eggs are also of interest, especially in those populations susceptible to developing macular degeneration and eye cataracts. SUMMARY: For these reasons, dietary recommendations aimed at restricting egg consumption should not be generalized to include all individuals. We need to acknowledge that diverse healthy populations experience no risk in developing coronary heart disease by increasing their intake of cholesterol but, in contrast, they may have multiple beneficial effects by the inclusion of eggs in their regular diet.

Kritchevsky SB. A review of scientific research and recommendations regarding eggs. J Am Coll Nutr. 2004 Dec;23(6 Suppl):596S-600S.

For much of the past 40 years, the public has been warned away from eggs because of a concern over coronary heart disease risk. This concern is based on three observations: 1. eggs are a rich source of dietary cholesterol; 2. when fed experimentally, dietary cholesterol increases serum cholesterol and; 3. high serum cholesterol predicts the onset of coronary heart disease. However, data from free-living populations show that egg consumption is not associated with higher cholesterol levels. Furthermore, as a whole, the epidemiologic literature does not support the idea that egg consumption is a risk factor for coronary disease. Within the nutritional community there is a growing appreciation that health derives from an overall pattern of diet rather than from the avoidance of particular foods, and there has been a shift in the tone in recent dietary recommendations away from "avoidance" messages to ones that promote healthy eating patterns. The most recent American Heart Association guidelines no longer include a recommendation to limit egg consumption, but recommend the adoption of eating practices associated with good health. Based on the epidemiologic evidence, there is no reason to think that such a healthy eating pattern could not include eggs.

Herron KL, Lofgren IE, Sharman M, Volek JS, Fernandez ML. Metabolism. 2004 Jun;53(6):823-30. High intake of cholesterol results in less atherogenic low-density lipoprotein particles in men and women independent of response classification.

The influence of a high-cholesterol diet on the atherogenicity of the low-density lipoprotein (LDL) particle was examined by measuring LDL peak diameter and composition, LDL susceptibility to oxidation, and the distribution of cholesterol between LDL subclasses. The crossover intervention randomly assigned 27 premenopausal women and 25 men (18 to 50 years) to an egg (640 mg/d additional dietary cholesterol) or placebo (0 mg/d additional dietary cholesterol) diet for 30 days, followed by a 3-week washout period. Subjects were classified as either hyperresponders (>2.5 mg/dL increase in plasma cholesterol for each 100 mg additional dietary cholesterol consumed) or hyporesponders to dietary cholesterol. Sex was found to have a significant effect on 3 of the parameters examined. LDL peak diameter was significantly larger (P <.005) in females (26.78 +/- 0.59 nm, n = 27) as compared with males (26.52 +/- 0.49 nm, n = 25), regardless of response to dietary cholesterol. The LDL particles of the male participants also had a higher number of triglyceride (TG) and cholesteryl ester (CE) molecules (P <.01); however, cholesterol ester transfer protein (CETP) activity was higher in females (P <.05). Response classification also revealed significant differences in the determination of LDL subclasses. Independent of sex, the LDL-1 particle (P <.05), which is considered to be less atherogenic, was predominant in hyperresponders and this finding was associated with increased cholesterol intake (interactive effect, P <.001). In addition, CETP and lecithin: cholesterol acyltransferase (LCAT) activities were higher in hyperresponders during the egg period (interactive effect, P <.05). Sex, response to cholesterol intake, and diet were not found to affect the susceptibility of LDL to oxidation (P > 0.5). Because LDL peak diameter was not decreased and the larger LDL-1 subclass was greater in hyperresponders following egg intake, these data indicate that the consumption of a high-cholesterol diet does not negatively influence the atherogenicity of the LDL particle.

Herron KL, Vega-Lopez S, Conde K, Ramjiganesh T, Shachter NS, Fernandez ML. Men classified as hypo- or hyperresponders to dietary cholesterol feeding exhibit differences in lipoprotein metabolism. J Nutr. 2003 Apr;133(4):1036-42.

The purpose of this study was to evaluate the differences that occur within the plasma compartment of normolipidemic men, classified on the basis of their response to prolonged consumption of additional dietary cholesterol. Using a crossover design, 40 men aged 18-57 y were randomly allocated to an egg (640 mg/d additional dietary cholesterol) or placebo group (0 mg/d additional dietary cholesterol), for two 30-d periods, which were separated by a 3-wk washout period. Subjects were classified as hypo- [increase in plasma total cholesterol (TC) of <0.05 mmol/L for each additional 100 mg of dietary cholesterol consumed] or hyperresponders (increase in TC of > or =0.06 mmol/L for each additional 100 mg of dietary cholesterol consumed) on the basis of their plasma reaction to the additional dietary cholesterol provided. Male hyporesponders did not experience an increase in LDL cholesterol (LDL-C) or HDL cholesterol (HDL-C) during the egg period, whereas both lipoproteins were significantly (P < 0.0001 and P < 0.05, respectively) elevated in hyperresponders. Although the LDL/HDL ratio was increased in male hyperresponders after the high cholesterol period, the mean increase experienced by this population was still within National Cholesterol Education Program guidelines. Furthermore, male hyperresponders had higher lecithin cholesterol acyltransferase (P < 0.05) and cholesteryl ester transfer protein (P < 0.05) activities during the egg period, which suggests an increase in reverse cholesterol transport. These data suggest that additional dietary cholesterol does not increase the risk of developing an atherogenic lipoprotein profile in healthy men, regardless of their response classification.

susanswan

What's wrong with just regular old eggs? They're really good for you. Excellent protein and healthy fats, lots of vitamins.

Stop obsessing over calories and concentrate on eating real food.

I am a fan of real food, too. Eggs are so cheap! I usually just mix up a bunch of whites with whatever number of yolks that make sense to me that day. Not a fan of just egg whites - unless we are talking meringue! Never tried egg beaters or cartoned fake egg products or whatever is in them. More chemicals. Just throw out the yolks and eat real eggs & slip yourself a yolk or two. As long as that isn't all you're eating!

joejccva71

Wait....

Mcrow are we talking about eating eggs or tipping over a bottle of crisco and drinking the entire bottle?

spa9177

I will never eat egg beaters...I do not substitute real for fake, to me it's not worth it. You only live once and I have a fried egg everyday with just a little butter so it doesn't stick. I have lost 37lbs. And I don't add any artifical flavorings...yuck yuck yuck.




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liftingbro

Not to beat the dead horse, but these were quotes that Alan Aragon used himself.

Fernandez ML. Dietary cholesterol provided by eggs and plasma lipoproteins in healthy populations. Curr Opin Clin Nutr Metab Care. 2006 Jan;9(1):8-12.

PURPOSE OF REVIEW: Extensive research has not clearly established a link between egg consumption and risk for coronary heart disease. The effects of egg intake on plasma lipids and low-density lipoprotein (LDL) atherogenicity in healthy populations need to be addressed. RECENT FINDINGS: The lack of connection between heart disease and egg intake could partially be explained by the fact that dietary cholesterol increases the concentrations of both circulating LDL and high-density lipoprotein (HDL) cholesterol in those individuals who experience an increase in plasma cholesterol following egg consumption (hyperresponders). It is also important to note that 70% of the population experiences a mild increase or no alterations in plasma cholesterol concentrations when challenged with high amounts of dietary cholesterol (hyporesponders). Egg intake has been shown to promote the formation of large LDL, in addition to shifting individuals from the LDL pattern B to pattern A, which is less atherogenic. Eggs are also good sources of antioxidants known to protect the eye; therefore, increased plasma concentrations of lutein and zeaxanthin in individuals consuming eggs are also of interest, especially in those populations susceptible to developing macular degeneration and eye cataracts. SUMMARY: For these reasons, dietary recommendations aimed at restricting egg consumption should not be generalized to include all individuals. We need to acknowledge that diverse healthy populations experience no risk in developing coronary heart disease by increasing their intake of cholesterol but, in contrast, they may have multiple beneficial effects by the inclusion of eggs in their regular diet.

Kritchevsky SB. A review of scientific research and recommendations regarding eggs. J Am Coll Nutr. 2004 Dec;23(6 Suppl):596S-600S.

For much of the past 40 years, the public has been warned away from eggs because of a concern over coronary heart disease risk. This concern is based on three observations: 1. eggs are a rich source of dietary cholesterol; 2. when fed experimentally, dietary cholesterol increases serum cholesterol and; 3. high serum cholesterol predicts the onset of coronary heart disease. However, data from free-living populations show that egg consumption is not associated with higher cholesterol levels. Furthermore, as a whole, the epidemiologic literature does not support the idea that egg consumption is a risk factor for coronary disease. Within the nutritional community there is a growing appreciation that health derives from an overall pattern of diet rather than from the avoidance of particular foods, and there has been a shift in the tone in recent dietary recommendations away from "avoidance" messages to ones that promote healthy eating patterns. The most recent American Heart Association guidelines no longer include a recommendation to limit egg consumption, but recommend the adoption of eating practices associated with good health. Based on the epidemiologic evidence, there is no reason to think that such a healthy eating pattern could not include eggs.

Herron KL, Lofgren IE, Sharman M, Volek JS, Fernandez ML. Metabolism. 2004 Jun;53(6):823-30. High intake of cholesterol results in less atherogenic low-density lipoprotein particles in men and women independent of response classification.

The influence of a high-cholesterol diet on the atherogenicity of the low-density lipoprotein (LDL) particle was examined by measuring LDL peak diameter and composition, LDL susceptibility to oxidation, and the distribution of cholesterol between LDL subclasses. The crossover intervention randomly assigned 27 premenopausal women and 25 men (18 to 50 years) to an egg (640 mg/d additional dietary cholesterol) or placebo (0 mg/d additional dietary cholesterol) diet for 30 days, followed by a 3-week washout period. Subjects were classified as either hyperresponders (>2.5 mg/dL increase in plasma cholesterol for each 100 mg additional dietary cholesterol consumed) or hyporesponders to dietary cholesterol. Sex was found to have a significant effect on 3 of the parameters examined. LDL peak diameter was significantly larger (P <.005) in females (26.78 +/- 0.59 nm, n = 27) as compared with males (26.52 +/- 0.49 nm, n = 25), regardless of response to dietary cholesterol. The LDL particles of the male participants also had a higher number of triglyceride (TG) and cholesteryl ester (CE) molecules (P <.01); however, cholesterol ester transfer protein (CETP) activity was higher in females (P <.05). Response classification also revealed significant differences in the determination of LDL subclasses. Independent of sex, the LDL-1 particle (P <.05), which is considered to be less atherogenic, was predominant in hyperresponders and this finding was associated with increased cholesterol intake (interactive effect, P <.001). In addition, CETP and lecithin: cholesterol acyltransferase (LCAT) activities were higher in hyperresponders during the egg period (interactive effect, P <.05). Sex, response to cholesterol intake, and diet were not found to affect the susceptibility of LDL to oxidation (P > 0.5). Because LDL peak diameter was not decreased and the larger LDL-1 subclass was greater in hyperresponders following egg intake, these data indicate that the consumption of a high-cholesterol diet does not negatively influence the atherogenicity of the LDL particle.

Herron KL, Vega-Lopez S, Conde K, Ramjiganesh T, Shachter NS, Fernandez ML. Men classified as hypo- or hyperresponders to dietary cholesterol feeding exhibit differences in lipoprotein metabolism. J Nutr. 2003 Apr;133(4):1036-42.

The purpose of this study was to evaluate the differences that occur within the plasma compartment of normolipidemic men, classified on the basis of their response to prolonged consumption of additional dietary cholesterol. Using a crossover design, 40 men aged 18-57 y were randomly allocated to an egg (640 mg/d additional dietary cholesterol) or placebo group (0 mg/d additional dietary cholesterol), for two 30-d periods, which were separated by a 3-wk washout period. Subjects were classified as hypo- [increase in plasma total cholesterol (TC) of <0.05 mmol/L for each additional 100 mg of dietary cholesterol consumed] or hyperresponders (increase in TC of > or =0.06 mmol/L for each additional 100 mg of dietary cholesterol consumed) on the basis of their plasma reaction to the additional dietary cholesterol provided. Male hyporesponders did not experience an increase in LDL cholesterol (LDL-C) or HDL cholesterol (HDL-C) during the egg period, whereas both lipoproteins were significantly (P < 0.0001 and P < 0.05, respectively) elevated in hyperresponders. Although the LDL/HDL ratio was increased in male hyperresponders after the high cholesterol period, the mean increase experienced by this population was still within National Cholesterol Education Program guidelines. Furthermore, male hyperresponders had higher lecithin cholesterol acyltransferase (P < 0.05) and cholesteryl ester transfer protein (P < 0.05) activities during the egg period, which suggests an increase in reverse cholesterol transport. These data suggest that additional dietary cholesterol does not increase the risk of developing an atherogenic lipoprotein profile in healthy men, regardless of their response classification.

Joe, all but one of those studies are talking about coronary disease and the connection to cholesterol. As I said earlier, cholesterol levels are not always an indicator of coronary diseas or health. In fact, those studies prove my point, increased cholesterol intake means increased serum cholesterol levels for most people. This is why I'm not too worried about people who have chronically high cholesterol levels inspite of diet changes and medications. It seems to me that some people can be perfectly healthy with high lipid levels. Like I said, I'm for eating eggs and don't find anything wrong with them though if you have a high cholesterol you should probably check with your MD in the case that you are one of those where cholesterol levels can affect health.

liftingbro

Wait....

Mcrow are we talking about eating eggs or tipping over a bottle of crisco and drinking the entire bottle?

Eggs are fine, as I said earier. The only time I would worry about eating eggs is if you have a high cholesterol already and have not been evaluated for coranary disease. If that's the case, you should at least have your lipids run and change your diet to see if your lpids also change. However, for most people I can't see where eating eggs regularly is a problem.

AdAstra47

Our bodies are funny things, all those hormones and chemicals interacting in unexpected ways, it makes it really hard to establish causality or figure out whether a study is useful or not...

Just from personal experience, I agree with those who say that a high percentage of fat or cholesterol in a food like eggs is not necessarily something to worry about. Fat and cholesterol are necessary for healthy cell function. It's not as if what you eat goes straight into your bloodstream as is; everything's digested & converted & processed first. Calories from carbs are far more likely to end up stored as body fat than calories from fat are! Weird, but true.

When I began seeing my current doctor, I had dangerously high cholesterol. He put me on a low-carb, high-protein diet. Those were my only two rules: stay under a certain number of carbs per day, and eat at least a certain number of protein grams per day. He left it entirely up to me, how to do that. I asked him about certain "bad" foods, or about avoiding certain foods to bring my cholesterol down, and he said I shouldn't worry about that. He said that losing weight overall, and eating fewer carbs, would take care of the cholesterol problem no matter what I ate. And four months later, here I am 30 lb lighter and my cholesterol levels are solidly in the middle of the healthy range across the board. So I don't know if that's true for everyone, but it's true for me.

So, again, I say, if you like the eggs, eat the eggs! :happy:

fattyfatfatfatyo

Love eggs. I eat two every morning with a few black beans, tomato, and Cholula.
Hard boiled eggs are the ultimate convenience food.

wildon883r

Two every day with a Pure protein bar for breakfast. This gives me the recommended 30G of protein within 30 minutes of waking recommened for weight loss. I'm good for 5 hours before i get hungry. Eggs are healthy, Cholesterol is not raised by eating eggs. Eggs are VERY healthy food. 70 calories per egg is really nothing. McDonalds sausage egg McMuffin has nearly 400 calories and is a pile of processed trash imo.

demonNIИ

I love eggs too!! Recently I switched back to Eggland's best to cut down on a bit of the cholesterol and sat fat

Melmade

I love them too. I might suggest a slice of really ripe avocado with cooked egg white, and then a whole egg in addition to that. I've been lately boiling eggs, eating one whole egg, and the other yolk I split between my son and my dog. Sounds kinda funny, but yolks have such great nutritive value for people and are really great for a dog's coat.

tigersword

I never understood eating just the egg whites. Egg whites are basically nutritionless. They contain about half of the egg's total protein, and really that's about it. The rest of the protein and just about all of the vitamins and minerals are in the yolk. Egg white isn't even a complete protein, so eating it by itself is useless.