Fatty liver
Replies
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I am not sure about fatty liver, but keto is good for fat burning, especially abdominal/visceral fat. I have lost 19.2kg and 20cm of my waist and 19cm from my hips in under 4 months. Within 3 months my triglycerides were down from 2 to 1.1and cholesterol down from 7 to 6.5.11
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I was diagnosed with a fatty liver maybe 4 tears ago. Was from poor diet doctor told me.0
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Do you have a doctor that specializes in livers?
It doesn’t seem that your doctor has told you much except to lose weight.3 -
milk thistle
11 -
TavistockToad wrote: »Saifnasirpk wrote: »TavistockToad wrote: »Saifnasirpk wrote: »goldthistime wrote: »Have you used MFP to set your goal calories and started tracking? I'd want to lose these last 5kgs in a slow and controlled fashion. Congrats on getting the first 6kgs off and cleaning up your diet.
I just started using MFP yesterday. I've been dieting 3 weeks now. What I did was cut in between snacks. Porridge for breakfast, a light lunch and a bowl of soup for dinner. You could say I was just having 2 meals a day. And I continued my exercises.
That doesn't sound like enough calories to be sustainable
Yes, I couldn't afford to lose weight slowly and gradually. I had to be more extreme because of the reason why I had to lose it. Basically I have to get low and check if my medical problem still persists.
Your doctor told you to crash diet to lose the weight quickly?
Rapid weightloss with a fatty liver is a bad idea. I read that it can lead to scarring.2 -
Fatty liver here too. Basically, I'm just trying to gradually lose weight by CICO. I've read lots of things online that may help. Turmeric helps the liver. Obviously, no alcohol. Exercise!! i'm 23 and overweight. My older brother is a normal weight, but has fatty liver. Must have been running in our family.2
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A ketogenic diet can be used effectively to reverse NAFLD. NAFLD is a form of insulin resistance. Like PCOS or dementia, high blood glucose is not always a sign of IR. A low carb diet reduces insulin which helps with treating NAFLD; a ketogenic diet, an even lower carb diet, may not even be needed.
https://www.ncbi.nlm.nih.gov/pubmed/19575599
http://www.diabesityinpractice.co.uk/media/content/_master/4311/files/pdf/dip4-3-102-8.pdf
Even small reductions in carbs, and increases in fats and proteins, seems to help NAFLD:
https://www.healio.com/hepatology/steatohepatitis-metabolic-liver-disease/news/online/{74b19e33-5565-42ce-b806-93fa61292657}/low-carb-diet-superior-to-low-fat-in-korean-patients-with-nafld
Exercise, weight loss, reduced stress and improved sleep has also been shown to help with insulin resistance in its many forms.
Good luck!2 -
Saifnasirpk wrote: »Over all I'm pretty fit. I exercise regularly playing Squash, Tennis and the occasionally running but I have some fat on my liver which doctors think is causing a raised ALT. I don't drink but I eat all sorts of junk food. So I've started dieting no junks. I've already lost 6kgs, gone from 86 to 80. I want to lose 5 more. My height is 6'3. Help
i will post this in hopes that people mind their own business and don't comment negatively on my post. i am so sick of all the naysayers on this board. but besides that, i don't know if this will apply to you, but i have been diagnosed with fatty liver and my boyfriend found a study that correlated fatty liver with a choline definciency in humans. something about how people with a choline defiency all got fatty liver and once they took some choline supplements the fatty liver resolved itself. i don't know if your fatty liver is caused by this, but the study gave people on tpn's (total parenteal nutrition or something) fed by a tube with a formula basically.. two formulas- one supplemented with choline and one not. the people who didn't get choline all developed fatty liver and the people who did get the choline did not. so i bought some alpha-gpc which is supposedly the best bio-available form of choline and took it for about 4 weeks. i forget the dosage and i haven't gotten feedback as the last time i went to the doctors i asked her to check my liver enzymes to see if it worked and she never did it. supposedly people who took the correct dosage for two weeks reversed their fatty liver. i can say i had much more energy while on the alph-gpc then before and felt less sluggish and more alert in general. i can't remember the dosage i took and threw the bottle away but it was the recommended amount on the bottle directions. (600mg maybe) so, maybe worth a try.. just thought i 'd mention it to you. i don't know if it worked, but i did take a whole bottles worth and had no ill effects that i could notice.4 -
alicebhsia wrote: »Saifnasirpk wrote: »Over all I'm pretty fit. I exercise regularly playing Squash, Tennis and the occasionally running but I have some fat on my liver which doctors think is causing a raised ALT. I don't drink but I eat all sorts of junk food. So I've started dieting no junks. I've already lost 6kgs, gone from 86 to 80. I want to lose 5 more. My height is 6'3. Help
i will post this in hopes that people mind their own business and don't comment negatively on my post. i am so sick of all the naysayers on this board. but besides that, i don't know if this will apply to you, but i have been diagnosed with fatty liver and my boyfriend found a study that correlated fatty liver with a choline definciency in humans. something about how people with a choline defiency all got fatty liver and once they took some choline supplements the fatty liver resolved itself. i don't know if your fatty liver is caused by this, but the study gave people on tpn's (total parenteal nutrition or something) fed by a tube with a formula basically.. two formulas- one supplemented with choline and one not. the people who didn't get choline all developed fatty liver and the people who did get the choline did not. so i bought some alpha-gpc which is supposedly the best bio-available form of choline and took it for about 4 weeks. i forget the dosage and i haven't gotten feedback as the last time i went to the doctors i asked her to check my liver enzymes to see if it worked and she never did it. supposedly people who took the correct dosage for two weeks reversed their fatty liver. i can say i had much more energy while on the alph-gpc then before and felt less sluggish and more alert in general. i can't remember the dosage i took and threw the bottle away but it was the recommended amount on the bottle directions. (600mg maybe) so, maybe worth a try.. just thought i 'd mention it to you. i don't know if it worked, but i did take a whole bottles worth and had no ill effects that i could notice.
Do you still have more energy?
Honestly, If something works, and appears to work, and isn't causing any harm, I'm not sure I'd quit. Or at least not quit until I got a positive response from my medical team.2 -
My mom recovered from fatty liver by reducing carbs and losing weight. She wasn't on a super low carb, just more carb conscious.3
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alicebhsia wrote: »Saifnasirpk wrote: »Over all I'm pretty fit. I exercise regularly playing Squash, Tennis and the occasionally running but I have some fat on my liver which doctors think is causing a raised ALT. I don't drink but I eat all sorts of junk food. So I've started dieting no junks. I've already lost 6kgs, gone from 86 to 80. I want to lose 5 more. My height is 6'3. Help
i will post this in hopes that people mind their own business and don't comment negatively on my post. i am so sick of all the naysayers on this board. but besides that, i don't know if this will apply to you, but i have been diagnosed with fatty liver and my boyfriend found a study that correlated fatty liver with a choline definciency in humans. something about how people with a choline defiency all got fatty liver and once they took some choline supplements the fatty liver resolved itself. i don't know if your fatty liver is caused by this, but the study gave people on tpn's (total parenteal nutrition or something) fed by a tube with a formula basically.. two formulas- one supplemented with choline and one not. the people who didn't get choline all developed fatty liver and the people who did get the choline did not. so i bought some alpha-gpc which is supposedly the best bio-available form of choline and took it for about 4 weeks. i forget the dosage and i haven't gotten feedback as the last time i went to the doctors i asked her to check my liver enzymes to see if it worked and she never did it. supposedly people who took the correct dosage for two weeks reversed their fatty liver. i can say i had much more energy while on the alph-gpc then before and felt less sluggish and more alert in general. i can't remember the dosage i took and threw the bottle away but it was the recommended amount on the bottle directions. (600mg maybe) so, maybe worth a try.. just thought i 'd mention it to you. i don't know if it worked, but i did take a whole bottles worth and had no ill effects that i could notice.
TPN is known to be hard on the liver, I'm not convinced this translates to an individual who is eating. I would be curious to see the study.4 -
My mom was diagnosed with non-alcoholic fatty liver several years ago. Admittedly, she has been overweight most of my life and isn't terribly physically fit. It wasn't until she was on a long-term corticosteroid for another unrelated health issue (autoimmune disorder) that her liver enzymes started to return to normal.1
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Fatty liver here too. Basically, I'm just trying to gradually lose weight by CICO. I've read lots of things online that may help. Turmeric helps the liver. Obviously, no alcohol. Exercise!! i'm 23 and overweight. My older brother is a normal weight, but has fatty liver. Must have been running in our family.
Hang in there buddy. We'll get over it
3 -
A ketogenic diet can be used effectively to reverse NAFLD. NAFLD is a form of insulin resistance. Like PCOS or dementia, high blood glucose is not always a sign of IR. A low carb diet reduces insulin which helps with treating NAFLD; a ketogenic diet, an even lower carb diet, may not even be needed.
https://www.ncbi.nlm.nih.gov/pubmed/19575599
http://www.diabesityinpractice.co.uk/media/content/_master/4311/files/pdf/dip4-3-102-8.pdf
Even small reductions in carbs, and increases in fats and proteins, seems to help NAFLD:
https://www.healio.com/hepatology/steatohepatitis-metabolic-liver-disease/news/online/{74b19e33-5565-42ce-b806-93fa61292657}/low-carb-diet-superior-to-low-fat-in-korean-patients-with-nafld
Exercise, weight loss, reduced stress and improved sleep has also been shown to help with insulin resistance in its many forms.
Good luck!
Thank you for the information1 -
StevefromMichigan wrote: »Saifnasirpk wrote: »StevefromMichigan wrote: »Did they diagnose with an MRI?
No, just an ultrasound. That's how he spotted the fatty liver
Ok. My thoughts are that you are already at/near an ideal body weight for a man your size. Usually, it would be recommended to diet when the patient is obese, which you do not appear to be. Considering where you are at in terms of weight now, you're probably not going to be able to lose too much more.
An ultrasound can pick up some things, but an MRI will show much more. Usually, if they find something on the ultrasound, an MRI is often recommended as the next step.
I recommend that you request to see a hepatologist. I believe the normal range for ALT is <40. You should make sure nothing else is going on here. It may just be fatty liver, but if it's something else, early treatment can be critical.
This is the best advice so far.
On diet, I'd continue reducing in a gradual fashion (my math has you at a BMI 22, but it wouldn't hurt to lose a bit more at a more gradual approach), and eat a traditionally healthful diet -- diets for non-alcoholic fatty liver often are lower fat and lower sugar/refined carbs, and recommend the usual vegetables, legumes, whole grains. But I would get it checked out very fully, as it seems weird, it's usually connected to obesity.4 -
Do you still eat high fructose corn syrup in your food? Cutting that out may help as it is a contributing factor to fatty liver.11
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kelly_stevens81 wrote: »Do you still eat high fructose corn syrup in your food? Cutting that out may help as it is a contributing factor to fatty liver.
How so?
*Edit- as compared to regular sugar/simple carbs, etc.1 -
It's probably a contributing factor only because it's in foods that are typically consumed without fiber or protein more often than not (especially soda). Not because all else equal it's that different to have a cookie with HFCS vs sucrose.
I personally would cut out soda, for sure (if OP consumes it, which he very well may not), and probably all sugary drinks, probably even juice, and eat fruit instead.3 -
My husband is in similar situation to OP. Doesn't drink... high ALT and has had an ultra sound scan. GP has also advised him to change his diet - very poor, take outs and junk food. He's due back at the GP next month so it will be interesting to see if change in diet has made any positive impact.
Interesting info re the choline study, not something that is checked for (deficiency in choline)0 -
MommyMeggo wrote: »kelly_stevens81 wrote: »Do you still eat high fructose corn syrup in your food? Cutting that out may help as it is a contributing factor to fatty liver.
How so?
*Edit- as compared to regular sugar/simple carbs, etc.
Fructose is metabolized by the liver. Too much may contribute to NAFLD.
https://www.ncbi.nlm.nih.gov/pubmed/23390127
"Dietary fructose in nonalcoholic fatty liver disease.
Abstract
Nonalcoholic fatty liver disease (NAFLD) is the most common chronic liver disease in adults and children. A number of genetic and environmental factors are known to predispose individuals to NAFLD. Certain dietary sugars, particularly fructose, are suspected to contribute to the development of NAFLD and its progression. The increasing quantity of fructose in the diet comes from sugar additives (most commonly sucrose and high fructose corn syrup) in beverages and processed foods. Substantial links have been demonstrated between increased fructose consumption and obesity, dyslipidemia, and insulin resistance. Growing evidence suggests that fructose contributes to the development and severity of NAFLD. In human studies, fructose is associated with increasing hepatic fat, inflammation, and possibly fibrosis. Whether fructose alone can cause NAFLD or if it serves only as a contributor when consumed excessively in the setting of insulin resistance, positive energy balance, and sedentary lifestyle is unknown. Sufficient evidence exists to support clinical recommendations that fructose intake be limited through decreasing foods and drinks high in added (fructose-containing) sugars."7 -
lemurcat12 wrote: »It's probably a contributing factor only because it's in foods that are typically consumed without fiber or protein more often than not (especially soda). Not because all else equal it's that different to have a cookie with HFCS vs sucrose.
I personally would cut out soda, for sure (if OP consumes it, which he very well may not), and probably all sugary drinks, probably even juice, and eat fruit instead.
Before I would drink lots of soda. But I'm about 4 weeks clean now.2 -
Enthusiast84 wrote: »My husband is in similar situation to OP. Doesn't drink... high ALT and has had an ultra sound scan. GP has also advised him to change his diet - very poor, take outs and junk food. He's due back at the GP next month so it will be interesting to see if change in diet has made any positive impact.
Interesting info re the choline study, not something that is checked for (deficiency in choline)
Good luck to him.1 -
MommyMeggo wrote: »kelly_stevens81 wrote: »Do you still eat high fructose corn syrup in your food? Cutting that out may help as it is a contributing factor to fatty liver.
How so?
*Edit- as compared to regular sugar/simple carbs, etc.
Fructose is metabolized by the liver. Too much may contribute to NAFLD.
https://www.ncbi.nlm.nih.gov/pubmed/23390127
"Dietary fructose in nonalcoholic fatty liver disease.
Abstract
Nonalcoholic fatty liver disease (NAFLD) is the most common chronic liver disease in adults and children. A number of genetic and environmental factors are known to predispose individuals to NAFLD. Certain dietary sugars, particularly fructose, are suspected to contribute to the development of NAFLD and its progression. The increasing quantity of fructose in the diet comes from sugar additives (most commonly sucrose and high fructose corn syrup) in beverages and processed foods. Substantial links have been demonstrated between increased fructose consumption and obesity, dyslipidemia, and insulin resistance. Growing evidence suggests that fructose contributes to the development and severity of NAFLD. In human studies, fructose is associated with increasing hepatic fat, inflammation, and possibly fibrosis. Whether fructose alone can cause NAFLD or if it serves only as a contributor when consumed excessively in the setting of insulin resistance, positive energy balance, and sedentary lifestyle is unknown. Sufficient evidence exists to support clinical recommendations that fructose intake be limited through decreasing foods and drinks high in added (fructose-containing) sugars."
This makes so much sense. I would drink a lot of coke.1 -
MommyMeggo wrote: »kelly_stevens81 wrote: »Do you still eat high fructose corn syrup in your food? Cutting that out may help as it is a contributing factor to fatty liver.
How so?
*Edit- as compared to regular sugar/simple carbs, etc.
Fructose is metabolized by the liver. Too much may contribute to NAFLD.
https://www.ncbi.nlm.nih.gov/pubmed/23390127
"Dietary fructose in nonalcoholic fatty liver disease.
Abstract
Nonalcoholic fatty liver disease (NAFLD) is the most common chronic liver disease in adults and children. A number of genetic and environmental factors are known to predispose individuals to NAFLD. Certain dietary sugars, particularly fructose, are suspected to contribute to the development of NAFLD and its progression. The increasing quantity of fructose in the diet comes from sugar additives (most commonly sucrose and high fructose corn syrup) in beverages and processed foods. Substantial links have been demonstrated between increased fructose consumption and obesity, dyslipidemia, and insulin resistance. Growing evidence suggests that fructose contributes to the development and severity of NAFLD. In human studies, fructose is associated with increasing hepatic fat, inflammation, and possibly fibrosis. Whether fructose alone can cause NAFLD or if it serves only as a contributor when consumed excessively in the setting of insulin resistance, positive energy balance, and sedentary lifestyle is unknown. Sufficient evidence exists to support clinical recommendations that fructose intake be limited through decreasing foods and drinks high in added (fr uctose-containing) sugars."
I'm not sure why this post got to two "Woo"s. The article that @nvmomketo shared was published in a respected, peer-reviewed medical journal. Perhaps MFP's meaning for "Woo" was misunderstood? It doesn't have a good connotation as in "who-hoo" but rather it refers to pseudoscience/unsubstantiated claims.0 -
MommyMeggo wrote: »kelly_stevens81 wrote: »Do you still eat high fructose corn syrup in your food? Cutting that out may help as it is a contributing factor to fatty liver.
How so?
*Edit- as compared to regular sugar/simple carbs, etc.
Fructose is metabolized by the liver. Too much may contribute to NAFLD.
https://www.ncbi.nlm.nih.gov/pubmed/23390127
"Dietary fructose in nonalcoholic fatty liver disease.
Abstract
Nonalcoholic fatty liver disease (NAFLD) is the most common chronic liver disease in adults and children. A number of genetic and environmental factors are known to predispose individuals to NAFLD. Certain dietary sugars, particularly fructose, are suspected to contribute to the development of NAFLD and its progression. The increasing quantity of fructose in the diet comes from sugar additives (most commonly sucrose and high fructose corn syrup) in beverages and processed foods. Substantial links have been demonstrated between increased fructose consumption and obesity, dyslipidemia, and insulin resistance. Growing evidence suggests that fructose contributes to the development and severity of NAFLD. In human studies, fructose is associated with increasing hepatic fat, inflammation, and possibly fibrosis. Whether fructose alone can cause NAFLD or if it serves only as a contributor when consumed excessively in the setting of insulin resistance, positive energy balance, and sedentary lifestyle is unknown. Sufficient evidence exists to support clinical recommendations that fructose intake be limited through decreasing foods and drinks high in added (fr uctose-containing) sugars."
I'm not sure why this post got to two "Woo"s. The article that @nvmomketo shared was published in a respected, peer-reviewed medical journal. Perhaps MFP's meaning for "Woo" was misunderstood? It doesn't have a good connotation as in "who-hoo" but rather it refers to pseudoscience/unsubstantiated claims.
Not sure either. I was thankful for the read. It makes sense.0 -
MommyMeggo wrote: »kelly_stevens81 wrote: »Do you still eat high fructose corn syrup in your food? Cutting that out may help as it is a contributing factor to fatty liver.
How so?
*Edit- as compared to regular sugar/simple carbs, etc.
Fructose is metabolized by the liver. Too much may contribute to NAFLD.
https://www.ncbi.nlm.nih.gov/pubmed/23390127
"Dietary fructose in nonalcoholic fatty liver disease.
Abstract
Nonalcoholic fatty liver disease (NAFLD) is the most common chronic liver disease in adults and children. A number of genetic and environmental factors are known to predispose individuals to NAFLD. Certain dietary sugars, particularly fructose, are suspected to contribute to the development of NAFLD and its progression. The increasing quantity of fructose in the diet comes from sugar additives (most commonly sucrose and high fructose corn syrup) in beverages and processed foods. Substantial links have been demonstrated between increased fructose consumption and obesity, dyslipidemia, and insulin resistance. Growing evidence suggests that fructose contributes to the development and severity of NAFLD. In human studies, fructose is associated with increasing hepatic fat, inflammation, and possibly fibrosis. Whether fructose alone can cause NAFLD or if it serves only as a contributor when consumed excessively in the setting of insulin resistance, positive energy balance, and sedentary lifestyle is unknown. Sufficient evidence exists to support clinical recommendations that fructose intake be limited through decreasing foods and drinks high in added (fr uctose-containing) sugars."
I'm not sure why this post got to two "Woo"s. The article that @nvmomketo shared was published in a respected, peer-reviewed medical journal. Perhaps MFP's meaning for "Woo" was misunderstood? It doesn't have a good connotation as in "who-hoo" but rather it refers to pseudoscience/unsubstantiated claims.
I didn't woo the post, but maybe because there's a common misunderstanding that HFCS has far more fructose than sugar or that fructose is always bad? Sucrose is about 50/50 glucose and fructose, and HFCS is about 45/55 glucose and fructose, and fruit also has fructose (in varying percentages). That's why -- as I explained above -- the bigger issue with HFCS is that it is likely more often consumed in circumstances where there is no protein or fiber to slow it down, so it has a bigger impact on the liver (on the converse, this is why fructose has less of an effect on blood sugar than glucose and does not lead to those spikes).
The message should NOT be that fructose is scary and should be avoided -- which has led to unfortunate demonizing of fruit at times -- but that excessive added sugar, especially when consumed without fiber or other foods, is a bad idea. This is why I think cutting back on soda and any sugary drinks is a great idea, with or without HFCS.6 -
MommyMeggo wrote: »kelly_stevens81 wrote: »Do you still eat high fructose corn syrup in your food? Cutting that out may help as it is a contributing factor to fatty liver.
How so?
*Edit- as compared to regular sugar/simple carbs, etc.
Fructose is metabolized by the liver. Too much may contribute to NAFLD.
https://www.ncbi.nlm.nih.gov/pubmed/23390127
"Dietary fructose in nonalcoholic fatty liver disease.
Abstract
Nonalcoholic fatty liver disease (NAFLD) is the most common chronic liver disease in adults and children. A number of genetic and environmental factors are known to predispose individuals to NAFLD. Certain dietary sugars, particularly fructose, are suspected to contribute to the development of NAFLD and its progression. The increasing quantity of fructose in the diet comes from sugar additives (most commonly sucrose and high fructose corn syrup) in beverages and processed foods. Substantial links have been demonstrated between increased fructose consumption and obesity, dyslipidemia, and insulin resistance. Growing evidence suggests that fructose contributes to the development and severity of NAFLD. In human studies, fructose is associated with increasing hepatic fat, inflammation, and possibly fibrosis. Whether fructose alone can cause NAFLD or if it serves only as a contributor when consumed excessively in the setting of insulin resistance, positive energy balance, and sedentary lifestyle is unknown. Sufficient evidence exists to support clinical recommendations that fructose intake be limited through decreasing foods and drinks high in added (fr uctose-containing) sugars."
I'm not sure why this post got to two "Woo"s. The article that @nvmomketo shared was published in a respected, peer-reviewed medical journal. Perhaps MFP's meaning for "Woo" was misunderstood? It doesn't have a good connotation as in "who-hoo" but rather it refers to pseudoscience/unsubstantiated claims.
Doesn't woo mean wow? Like Facebooks wow icon. I found this I information to be most useful as I used to drink a lot of soda and juices. Very helpful0 -
Saifnasirpk wrote: »MommyMeggo wrote: »kelly_stevens81 wrote: »Do you still eat high fructose corn syrup in your food? Cutting that out may help as it is a contributing factor to fatty liver.
How so?
*Edit- as compared to regular sugar/simple carbs, etc.
Fructose is metabolized by the liver. Too much may contribute to NAFLD.
https://www.ncbi.nlm.nih.gov/pubmed/23390127
"Dietary fructose in nonalcoholic fatty liver disease.
Abstract
Nonalcoholic fatty liver disease (NAFLD) is the most common chronic liver disease in adults and children. A number of genetic and environmental factors are known to predispose individuals to NAFLD. Certain dietary sugars, particularly fructose, are suspected to contribute to the development of NAFLD and its progression. The increasing quantity of fructose in the diet comes from sugar additives (most commonly sucrose and high fructose corn syrup) in beverages and processed foods. Substantial links have been demonstrated between increased fructose consumption and obesity, dyslipidemia, and insulin resistance. Growing evidence suggests that fructose contributes to the development and severity of NAFLD. In human studies, fructose is associated with increasing hepatic fat, inflammation, and possibly fibrosis. Whether fructose alone can cause NAFLD or if it serves only as a contributor when consumed excessively in the setting of insulin resistance, positive energy balance, and sedentary lifestyle is unknown. Sufficient evidence exists to support clinical recommendations that fructose intake be limited through decreasing foods and drinks high in added (fr uctose-containing) sugars."
I'm not sure why this post got to two "Woo"s. The article that @nvmomketo shared was published in a respected, peer-reviewed medical journal. Perhaps MFP's meaning for "Woo" was misunderstood? It doesn't have a good connotation as in "who-hoo" but rather it refers to pseudoscience/unsubstantiated claims.
Doesn't woo mean wow? Like Facebooks wow icon. I found this I information to be most useful as I used to drink a lot of soda and juices. Very helpful
Not on MFP. Many people, including myself, are frustrated by this relatively new icon. In the MFP world, it does not mean "wow" or have another positive connotation. It's very confusing, which is why I made my post. I did not want MFP readers to think that the article posted and discussed was pseudoscience.2 -
Ugh, my doctor just told me I had a fatty liver. I found this interesting.2
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Saifnasirpk wrote: »MommyMeggo wrote: »kelly_stevens81 wrote: »Do you still eat high fructose corn syrup in your food? Cutting that out may help as it is a contributing factor to fatty liver.
How so?
*Edit- as compared to regular sugar/simple carbs, etc.
Fructose is metabolized by the liver. Too much may contribute to NAFLD.
https://www.ncbi.nlm.nih.gov/pubmed/23390127
"Dietary fructose in nonalcoholic fatty liver disease.
Abstract
Nonalcoholic fatty liver disease (NAFLD) is the most common chronic liver disease in adults and children. A number of genetic and environmental factors are known to predispose individuals to NAFLD. Certain dietary sugars, particularly fructose, are suspected to contribute to the development of NAFLD and its progression. The increasing quantity of fructose in the diet comes from sugar additives (most commonly sucrose and high fructose corn syrup) in beverages and processed foods. Substantial links have been demonstrated between increased fructose consumption and obesity, dyslipidemia, and insulin resistance. Growing evidence suggests that fructose contributes to the development and severity of NAFLD. In human studies, fructose is associated with increasing hepatic fat, inflammation, and possibly fibrosis. Whether fructose alone can cause NAFLD or if it serves only as a contributor when consumed excessively in the setting of insulin resistance, positive energy balance, and sedentary lifestyle is unknown. Sufficient evidence exists to support clinical recommendations that fructose intake be limited through decreasing foods and drinks high in added (fr uctose-containing) sugars."
I'm not sure why this post got to two "Woo"s. The article that @nvmomketo shared was published in a respected, peer-reviewed medical journal. Perhaps MFP's meaning for "Woo" was misunderstood? It doesn't have a good connotation as in "who-hoo" but rather it refers to pseudoscience/unsubstantiated claims.
Doesn't woo mean wow? Like Facebooks wow icon. I found this I information to be most useful as I used to drink a lot of soda and juices. Very helpful
Not on MFP. Many people, including myself, are frustrated by this relatively new icon. In the MFP world, it does not mean "wow" or have another positive connotation. It's very confusing, which is why I made my post. I did not want MFP readers to think that the article posted and discussed was pseudoscience.
It was originally intended to have a dual meaning (one positive, one not), and still gets used positively in some sections on MFP and by some users. You simply cannot presume to know what anyone meant by "woo" in a particular case.
That said, I read the post as suggesting there was a meaningful difference between HFCS and sugar all else equal, and that's not accurate. I'll note that the link wasn't specifically about HFCS, but it was supplied as alleged support for the idea that OP should cut out HFCS as the culprit.
(I do, as stated above, think cutting out certain HFCS containing foods and limiting others is a good idea, but would not consider HFCS different in that respect from added sugar or even fruit juice, if OP hypothetically consumed a lot of that.)1
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